Tuberc Respir Dis > Volume 53(6); 2002 > Article
Tuberculosis and Respiratory Diseases 2002;53(6):595-611.
DOI: https://doi.org/10.4046/trd.2002.53.6.595    Published online December 1, 2002.
Molecular Mechanisms of Neutrophil Activation in Acute Lung Injury.
Ho Kee Yum
Department of Internal Medicine, School of Medicine, Inje University, Seoul, Korea. pulho@korea.com
Abstract
ABBREVIATIONS: Akt/PKB protein kinase B, ALI acute lung injury, ARDS acute respiratory distress syndrome, CREB C-AMP response element binding protein, ERK extracelluar signal-related kinase, fMLP fMet-Leu-Phe, G-CSF granulocyte colony-stimulating factor, IL interleukin, ILK integrin-linked kinase, JNK Jun N-terminal kinase, LPS lipopolysaccharide, MAP mitogen-activated protein, MEK MAP/ERK kinase, MIP-2 macrophage inflammatory protein-2, MMP matrix metalloproteinase, MPO myeloperoxidase, NADPH nicotinamide adenine dinucleotide phosphate, NE neutrophil elastase, NF-kappaB nuclear factor-kappa B, NOS nitric oxide synthase, p38 MAPK p38 mitogen activated protein kinase, PAF platelet activating factor, PAKs p21-activated kinases, PMN polymorphonuclear leukocytes, PI3-K phosphatidylinositol 3-kinase, PyK proline-rich tyrosine kinase, ROS reactive oxygen species, TNF-alpha tumor necrosis factor-alpha.
Key Words: Acute lung injury, Neutrophil, Signal transduction


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