Adrenal Cortical Function in Pulmonary Tuberculosis

Kim, Sun Taek; Kim, Kyung Sik

doi:1961.0.10.85

Tuberc Respir Dis > Issue 10; 1961 > Article

Tuberculosis and Respiratory Diseases 1961;10:85-89.
DOI: https://doi.org/10.4046/trd.1961.10.1.85 Published online December 1, 1961.

Adrenal Cortical Function in Pulmonary Tuberculosis

Sun Taek Kim, Kyung Sik Kim

Department of Internal Medicine, Medical College Seoul National University, Seoul, Korea

폐결핵환자의 부신피질기능에 관하여

김순택, 김경식

Abstract

We measured total 17-Hydroxycorticosteroid excretion and its increasing rate after ACTH administration by using Porter-Silber method and 17-ketosteroid excretion by using Holtorff and Koch’s method. and had measured reducing rate of circulating eosinophile count after ACTH administration in order to investigate adrenal cortical function of the pulmonary tuberculosis. 1) In Normal Control group. 17. Hydroxycorticosteroid excretion 4. 91-7 93mg average 5. 46mg Increasing ratio of 17-Hydroxycorticosteroid after ACTH administration 30.89% 17-Ketosteroid excretion 13. 66-23. 91mg average 17.71ing Reducing ratio of eosinophile count after ACTH administration 64%. In Minimal Tuberculosis group 17-Hydroxycorticosteroid excretion 1. 93-4. 30mg average 3. 19m9 17-Ketosteroid excretion 5.42-13. 30mg average 10. 36mg Reducing ratio of eosinophile count after ACTH administration 44. 83% In far advanced group 17-Hydroxycorticosteroid excretion 0. 83-4. 43mg average 2. 46mg Increasing ratio of 17-Hydroxycorticosteroid after ACTH administration 196. 7% 17-Ketosteroid excretion 4.60-14. 43mg average 7. 67mg Reducing ratio of eosinophile count after ACTH administration 52% (8 hrs I. V. method) 2) The adrenal cortical function of the pulmonary tuberculosis depressed and its degree was almost parallel with severity of the diseases.

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